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Volume 10, Number 4—April 2004

Research

Maternal Malaria and Perinatal HIV Transmission, Western Kenya1,2

John G. Ayisi*†, Anna M. van Eijk*†, Robert D. Newman‡Comments to Author , Feiko O. ter Kuile*†‡, Ya Ping Shi*‡, Chunfu Yang‡, Margarette S. Kolczak‡, Juliana A. Otieno§, Ambrose O. Misore§, Piet A. Kager†, Renu B. Lal‡, Richard W. Steketee‡, and Bernard L. Nahlen‡¶
Author affiliations: *Centre for Vector Biology and Control Research, Kenya Medical Research Institute, Kisumu, Kenya; †University of Amsterdam, Amsterdam, The Netherlands; ‡Centers for Disease Control and Prevention, Atlanta, Georgia, USA; §Ministry of Health, Kisumu, Kenya; ¶World Health Organization, Geneva, Switzerland

Main Article

Figure 1

The effect of viral load and placental malaria density on risk for perinatal HIV transmission, western Kenya, 1996–2001. Women with low- (<10,000 parasites/μL, circles) and high- (>10,000 parasites/μL, squares) density placental malaria are compared with women without placental malaria (represented by the horizontal dashed line). RR, relative risk. Error bars refer to 95% confidence interval.

Figure 1. The effect of viral load and placental malaria density on risk for perinatal HIV transmission, western Kenya, 1996–2001. Women with low- (<10,000 parasites/μL, circles) and high- (>10,000 parasites/μL, squares) density placental malaria are compared with women without placental malaria (represented by the horizontal dashed line). RR, relative risk. Error bars refer to 95% confidence interval.

Main Article

1This work was presented in part at the Epidemic Intelligence Service Conference, Centers for Disease Control and Prevention, April 2001, Atlanta, GA.

2Use of trade names is for identification only and does not imply endorsement by the Kenya Medical Research Institute or The Ministry of Health, Kenya, or by the Public Health Service, U.S. Department of Health and Human Services.

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