Emerging Infectious Disease ISSN: 1080-6059

Rickettsia felis Infection, Tunisia

Abir Znazen*†, Jean-Marc Rolain*, Nader Hammami†, Adnane Hammami†, Mounir Ben Jemaa‡, and Didier Raoult*

Author affiliations: *Université de la Méditerranée, Marseille, France; †Universitaire Habib Bourguiba, Sfax, Tunisia; ‡Universitaire Hédi Chaker, Sfax, Tunisia

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Figure

Pictures of immunofluorescence assay performed on serum specimens with proven Rickettsia conorii (A), R. felis (B), or R. typhi (C) infection showing cross-reactive antibodies. Antigens tested were R. conorii (column 1), R. felis (column 2), and R. typhi (column 3). The serum with R. conorii infection reacts with R. conorii and R. felis antigens but not with R. typhi (A). Conversely, the serum with R. typhi infection reacts with R. typhi and R. felis but not with R. conorii (C). Finally, the ser

Figure. Pictures of immunofluorescence assay performed on serum specimens with proven Rickettsia conorii (A), R. felis (B), or R. typhi (C) infection showing cross-reactive antibodies. Antigens tested were R. conorii (column 1), R. felis (column 2), and R. typhi (column 3). The serum with R. conorii infection reacts with R. conorii and R. felis antigens but not with R. typhi (A). Conversely, the serum with R. typhi infection reacts with R. typhi and R. felis but not with R. conorii (C). Finally, the serum with R. felis infection reacts with R. felis, R. conorii, and R. typhi.

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