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Volume 15, Number 9—September 2009

Research

Genetics and Pathogenesis of Feline Infectious Peritonitis Virus

Meredith A. BrownComments to Author , Jennifer L. Troyer, Jill Pecon-Slattery, Melody E. Roelke, and Stephen J. O’Brien
Author affiliations: National Cancer Institute, Frederick, Maryland, USA (M.A. Brown, J. Pecon-Slattery, S.J. O’Brien); SAIC-Frederick, Inc., Frederick (J.L. Troyer, M.E. Roelke)

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Figure 3

A) Feline coronavirus genome indicating PCR products obtained (bars). Structural proteins are shaded in dark gray; nonstructural proteins are shaded in light gray. B) Forward and reverse primers used to amplify virus segments are listed in 5′ → 3′ orientation. The number of source cats and cloned sequences generated from feline infectious peritonitis (FIP) cases and feline enteric coronavirus (FECV) asymptomatic cats are presented. Pol, polymerase; NSP, nonstructural protein; FIPV, feline infect

Figure 3. A) Feline coronavirus genome indicating PCR products obtained (bars). Structural proteins are shaded in dark gray; nonstructural proteins are shaded in light gray. B) Forward and reverse primers used to amplify virus segments are listed in 5′ → 3′ orientation. The number of source cats and cloned sequences generated from feline infectious peritonitis (FIP) cases and feline enteric coronavirus (FECV) asymptomatic cats are presented. Pol, polymerase; NSP, nonstructural protein; FIPV, feline infectious peritonitis virus.

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