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Volume 16, Number 3—March 2010
Research

Use of Avian Bornavirus Isolates to Induce Proventricular Dilatation Disease in Conures

Patricia Gray, Sharman Hoppes, Paulette Suchodolski, Negin Mirhosseini, Susan Payne, Itamar Villanueva, H.L Shivaprasad, Kirsi S. Honkavuori, Thomas Briese, Sanjay M. Reddy, and Ian TizardComments to Author 
Author affiliations: Texas A&M University, College Station, Texas, USA (P. Gray, S. Hoppes, P. Suchodolski, N. Mirhosseini, S. Payne, I. Villanueva, S.M. Reddy, I. Tizard); California Animal Health and Food Safety Laboratory System, Fresno, California, USA (H.L. Shivaprasad); Columbia University, New York, New York, USA (K.S. Honkavuori, T. Briese)

Main Article

Figure 5

PCR of avian bornavirus N-protein in different areas of the brains of A) 2 Patagonian conures (PG7 and PG8) inoculated 55 days earlier with avian bornavirus–infected duck embryonic fibroblasts and B) control, uninfected bird, PG5. HB, hindbrain; FB, forebrain; MB, midbrain; Cereb., cerebrum.

Figure 5. PCR of avian bornavirus N-protein in different areas of the brains of A) 2 Patagonian conures (PG7 and PG8) inoculated 55 days earlier with avian bornavirus–infected duck embryonic fibroblasts and B) control, uninfected bird, PG5. HB, hindbrain; FB, forebrain; MB, midbrain; Cereb., cerebrum.

Main Article

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Page updated: December 14, 2010
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The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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