Use of Avian Bornavirus Isolates to Induce Proventricular Dilatation Disease in Conures
Patricia Gray, Sharman Hoppes, Paulette Suchodolski, Negin Mirhosseini, Susan Payne, Itamar Villanueva, H.L Shivaprasad, Kirsi S. Honkavuori, Thomas Briese, Sanjay M. Reddy, and Ian Tizard
Author affiliations: Texas A&M University, College Station, Texas, USA (P. Gray, S. Hoppes, P. Suchodolski, N. Mirhosseini, S. Payne, I. Villanueva, S.M. Reddy, I. Tizard); California Animal Health and Food Safety Laboratory System, Fresno, California, USA (H.L. Shivaprasad); Columbia University, New York, New York, USA (K.S. Honkavuori, T. Briese)
Figure 5. PCR of avian bornavirus N-protein in different areas of the brains of A) 2 Patagonian conures (PG7 and PG8) inoculated 55 days earlier with avian bornavirus–infected duck embryonic fibroblasts and B) control, uninfected bird, PG5. HB, hindbrain; FB, forebrain; MB, midbrain; Cereb., cerebrum.
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