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Volume 17, Number 2—February 2011

Letter

Maternal–Fetal Transmission of Cryptococcus gattii in Harbor Porpoise

Stephanie A. NormanComments to Author , Stephen Raverty, Erin Zabek, Sandra Etheridge, John K.B. Ford, Linda M.N. Hoang, and Muhammad Morshed
Author affiliations: Author affiliations: Marine-Med: Marine Research, Epidemiology, and Veterinary Medicine, Bothell, Washington, USA (S.A. Norman); Central Puget Sound Marine Mammal Stranding Network, Greenbank, Washington, USA (S.A. Norman); British Columbia Ministry of Agriculture and Lands, Abbotsford, British Columbia, Canada (S. Raverty, E. Zabek, S. Etheridge); Fisheries and Oceans, Canada, Nanaimo, British Columbia, Canada (J.K.B. Ford); British Columbia Center for Disease Control, Vancouver, British Columbia, Canada (L.M.N. Hoang, M. Morshed); University of British Columbia, Vancouver (L.M.N. Hoang, M. Morshed)

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Figure

A. A) Enlarged mediastinal lymph nodes of a stranded, pregnant, harbor porpoise (Phocoena phocoena) infected with Cryptococcus gattii that was transmitted to its fetus. B) Mucicarmine–stained sections of fetal mediastinal lymph node, showing C. gattii extracellular yeast aggregates (original magnification ×20). Scale bar = 50 μm.

Figure. A. A) Enlarged mediastinal lymph nodes of a stranded, pregnant, harbor porpoise (Phocoena phocoena) infected with Cryptococcus gattii that was transmitted to its fetus. B) Mucicarmine–stained sections of fetal mediastinal lymph node, showing C. gattii extracellular yeast aggregates (original magnification ×20). Scale bar = 50 μm.

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