Emerging Infectious Disease ISSN: 1080-6059

Deaths Associated with Human Adenovirus-14p1 Infections, Europe, 2009–2010

Michael J. Carr

, Adriana E. Kajon, Xiaoyan Lu, Linda Dunford, Paul O’Reilly, Paul Holder, Cillian F. De Gascun, Suzie Coughlan, Jeff Connell, Dean D. Erdman, and William W. Hall

Author affiliations: Author affiliations: National Virus Reference Laboratory–University College Dublin, Dublin, Ireland (M.J. Carr, L. Dunford, P. O’Reilly, P. Holder, C.F. De Gascun, S. Coughlan, J. Connell, W.W. Hall); Lovelace Respiratory Research Institute, Albuquerque, New Mexico, USA (A.E. Kajon); Centers for Disease Control and Prevention, Atlanta, Georgia, USA (X. Lu, D.D. Erdman)

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Figure 1

Maximum-likelihood trees of the full-length fiber (A), E1A (B), and hexon (C) open reading frames of adenovirus B2 subgenera. Phylogenetic analysis was performed by using reference sequences from GenBank for the adenovirus B2 subgenera, including prototype reference strains. The query sequences from this study are identical and are represented in boldface. The tree was built in PAUP* (23) on the basis of the HKY85 model of evolution and for the fiber tree also with a β distribution and used midp

Figure 1. Maximum-likelihood trees of the full-length fiber (A), E1A (B), and hexon (C) open reading frames of adenovirus B2 subgenera. Phylogenetic analysis was performed by using reference sequences from GenBank for the adenovirus B2 subgenera, including prototype reference strains. The query sequences from this study are identical and are represented in boldface. The tree was built in PAUP* (23) on the basis of the HKY85 model of evolution and for the fiber tree also with a β distribution and used midpoint rooting. Bootstrap resampling (n = 1,000) was performed by using the neighbor-joining algorithm. Scale bars indicate nucleotide substitutions per site.

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