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Volume 18, Number 2—February 2012

Research

Pathogenesis of Avian Bornavirus in Experimentally Infected Cockatiels

Anne K. PiepenbringComments to Author , Dirk Enderlein, Sibylle Herzog, Erhard F. Kaleta, Ursula Heffels-Redmann, Saskia Ressmeyer, Christiane Herden, and Michael Lierz
Author affiliations: Justus Liebig University Giessen, Giessen, Germany

Main Article

Figure 1

Timing of the first detection of avian bornavirus (ABV) RNA in cockatiels that had been intracerebrally or intravenously inoculated with ABV. ABV RNA was amplified significantly earlier in samples from intracerebrally inoculated birds compared with intravenously inoculated birds (α = 0.05 by using the Wilcoxon-Mann-Whitney test). A noninoculated sentinel bird, which was housed with the intracerebrally inoculated group of cockatiels, was the last bird to shed ABV RNA.

Figure 1. Timing of the first detection of avian bornavirus (ABV) RNA in cockatiels that had been intracerebrally or intravenously inoculated with ABV. ABV RNA was amplified significantly earlier in samples from intracerebrally inoculated birds compared with intravenously inoculated birds (α = 0.005 by using the Wilcoxon-Mann-Whitney test). A noninoculated sentinel bird, which was housed with the intracerebrally inoculated group of cockatiels, was the last bird to shed ABV RNA.

Main Article

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