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Volume 20, Number 12—December 2014
Research

Geographic Divergence of Bovine and Human Shiga Toxin–Producing Escherichia coli O157:H7 Genotypes, New Zealand1

Patricia JarosComments to Author , Adrian L. Cookson, Donald M. Campbell, Gail E. Duncan, Deborah Prattley, Philip E. Carter, Thomas E. Besser, Smriti Shringi, Steve Hathaway, Jonathan C. Marshall, and Nigel P. French
Author affiliations: Massey University, Palmerston North, New Zealand (P. Jaros, D. Prattley, J.C. Marshall, N.P. French); AgResearch Ltd, Palmerston North (A.L. Cookson); Ministry for Primary Industries, Wellington, New Zealand (D.M. Campbell, G.E. Duncan, S. Hathaway); Institute of Environmental Science and Research Ltd, Porirua, New Zealand (P. Carter); Washington State University, Pullman, Washington, USA (T.E. Besser, S. Shringi)

Main Article

Figure 1

Proportional distributions, stratified by year, of Shiga toxin–encoding bacteriophage insertion (SBI) types AY2a, WY12a, and ASY2c/SY2c of 363 human Shiga toxin–producing Escherichia coli O157:H7 isolates from clinical case-patients in New Zealand, 2008–2011. Error bars indicate 95% CIs.

Figure 1. Proportional distributions, stratified by year, of Shiga toxin–encoding bacteriophage insertion (SBI) types AY2a, WY12a, and ASY2c/SY2c of 363 human Shiga toxin–producing Escherichia coli O157:H7 isolates from clinical case-patients in New Zealand, 2008–2011. Error bars indicate 95% CIs.

Main Article

1Preliminary results from this study were presented at the New Zealand Veterinary Association Conference; June 16–20, 2014, Hamilton, New Zealand.

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Page updated: November 18, 2014
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The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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