Du-Ping Zheng*1, Teryl K. Frey* , Joseph Icenogle†, Shigetaka Katow†‡, Emily S. Abernathy*†, Ki-Joon Song§, Wen-Bo Xu¶, Vitaly Yarulin#, R.G. Desjatskova#, Yair Aboudy**, Gisela Enders††, and Margaret Croxson‡‡
Author affiliations: *Georgia State University, Atlanta, Georgia, USA; †Centers for Disease Control and Prevention, Atlanta, Georgia, USA; ‡National Institute of Infectious Diseases, Tokyo, Japan; §Korea University, Seoul, Korea; ¶Chinese Centers for Disease Control and Prevention, Beijing, China; #Institute of Viral Preparations, Moscow, Russia; **Chaim Sheba Medical Center, Tel-Hashomer, Israel; ††Institute for Virology, Infectiology and Epidemiology, Stuttgart, Germany; ‡‡Auckland Hospital, Auckland, New Zealand
Figure 2. Phylogenetic trees. Trees were made by the maximum likelihood method in the Tree-Puzzle 5.0 program (1,000 puzzling steps) using the 5′ half (640 nt: 8291–8930) or 3′ half (539 nt: 8931–9469) of the E1 gene sequence. Bootstrapping values (out of 100) for each node are given.
1Current address: Respiratory and Enteric Viruses Branch, Division of Viral and Rickettsial Diseases, National Center for Infectious Diseases, Centers for Disease Control and Prevention, Atlanta, GA, USA.
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