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Volume 13, Number 12—December 2007
Dispatch

WU Polyomavirus in Children, Canada

Yacine Abed*, David Wang†, and Guy Boivin*Comments to Author 
Author affiliations: *Centre Hospitalier Universitaire de Québec and Research Center in Infectious Diseases, Quebec City, Quebec, Canada; †Washington University School of Medicine, St. Louis, Missouri, USA;

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Figure

Phylogenetic analysis of Canadian WU polyomavirus strains CanWUT9–2003, CanWUT38–2003, CanWUC40–2003 and CanWUT52–2003 (shown in boldface), based on nucleotide sequences of the VP2 region. Multiple nucleotide sequence alignments were performed by using the ClustalW program and a phylogenetic tree was constructed with the MEGA 3.1 software using the neighbor-joining algorithm with Kimura-2 parameters (9). The analysis included WU strains previously identified from Australian and American cohorts (7) i.e., B9, S6, B28, B37, B22, B24, B35, B10, B1, B17, B0, B12, B20, B14, B34, B21, B3, and B2 (GenBank accession nos.: EF444592, EF444593, EF444590, EF444589, EF444588, EF444587, EF444586, EF444584, EF444583, EF444582, EF-444555, EF444557, EF444561, EF444562, EF444572, EF444585, EF444567, and EF444591, respectively).

Figure. Phylogenetic analysis of Canadian WU polyomavirus strains CanWUT9–2003, CanWUT38–2003, CanWUC40–2003 and CanWUT52–2003 (shown in boldface), based on nucleotide sequences of the VP2 region. Multiple nucleotide sequence alignments were performed by using the ClustalW program and a phylogenetic tree was constructed with the MEGA 3.1 software using the neighbor-joining algorithm with Kimura-2 parameters (9). The analysis included WU strains previously identified from Australian and American cohorts (7) i.e., B9, S6, B28, B37, B22, B24, B35, B10, B1, B17, B0, B12, B20, B14, B34, B21, B3, and B2 (GenBank accession nos.: EF444592, EF444593, EF444590, EF444589, EF444588, EF444587, EF444586, EF444584, EF444583, EF444582, EF-444555, EF444557, EF444561, EF444562, EF444572, EF444585, EF444567, and EF444591, respectively).

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