Volume 10, Number 10—October 2004
Dispatch
Emerging Enteropathogenic Escherichia coli Strains?
Tânia A. Tardelli Gomes*
, Kinue Irino†, Dennys M. Girão*‡§¶, Valéria BC Girão¶, Beatriz EC Guth*, Tânia MI Vaz*†, Fabiana C Moreira*, Silvia H Chinarelli#, and Mônica AM Vieira*
, Kinue Irino†, Dennys M. Girão*‡§¶, Valéria BC Girão¶, Beatriz EC Guth*, Tânia MI Vaz*†, Fabiana C Moreira*, Silvia H Chinarelli#, and Mônica AM Vieira*
Table 2
Prevalence of distinct combinations of virulence-related DNA sequences in eae+ EAF- stx- Escherichia coli strains outside the EPEC serogroups in three cities in Brazila
| Genetic profileb | No. (%) of strains |
|||
|---|---|---|---|---|
| Total (n = 99) | São Paulo (n = 59) | Rio de Janeiro (n = 23) | Ribeirão Preto (n = 17) | |
| eae | 31 (31.1) | 19 (32.2) | 5 (21.8) | 7 (41.1) |
| eae hly astA pet irp2 | 8 (8.1) | 8 (13.6) | 0 | 0 |
| eae hly | 6 (6.1) | 5 (8.5) | 0 | 1(5.9) |
| eae shf | 5 (5.1) | 1(1.7) | 3 (13.1) | 1(5.9) |
| eae irp2 | 5(5.1) | 4 (6.8) | 1(4.3) | 0 |
| eae perA bfpA astA | 4 (4.0) | 1(1.7) | 3 (13.1) | 0 |
| eae perA bfpA | 4 (4.0) | 0 | 4 (17.4) | 0 |
| eae hly daaC afa astA pet irp2 | 3 (3.0) | 3 (5.1) | 0 | 0 |
| eae perA | 3 (3.0) | 0 | 0 | 3 (17.6) |
| eae perA hly astA pet irp2 | 2 (2.0) | 1(1.7) | 0 | 1(5.9) |
| eae EHEC-hly astA | 2 (2.0) | 2 (3.4) | 0 | 0 |
| eae astA irp2 | 2 (2.0) | 2(3.4) | 0 | 0 |
| eae bfpA | 2 (2.0) | 1(1.7) | 1(4.3) | 0 |
| eae EHEC-hly | 2 (2.0) | 0 | 2 (8.7) | 0 |
| eae hly daaC afa pap sfa astA shf pet irp2 | 1(1.0) | 1(1.7) | 0 | 0 |
| eae hly daaC afa shf irp2 | 1(1.0) | 1(1.7) | 0 | 0 |
| eae perA bfpA hly pet | 1 (1.0) | 0 | 0 | 1(5.9) |
| eae perA hly daaC afa | 1(1.0) | 0 | 0 | 1(5.9) |
| eae perA bfpA astA irp2 | 1(1.0) | 1(1.7) | 0 | 0 |
| eae hly pap afa irp2 | 1(1.0) | 0 | 1(4.3) | 0 |
| eae hly daaC afa astA | 1(1.0) | 1(1.7) | 0 | 0 |
| eae hly astA shf irp2 | 1(1.0) | 1(1.7) | 0 | 0 |
| eae perA bfpA hly | 1(1.0) | 0 | 0 | 1(5.9) |
| eae hly astA irp2 | 1(1.0) | 1(1.7) | 0 | 0 |
| eae hly shf irp2 | 1(1.0) | 1(1.7) | 0 | 0 |
| eae perA astA | 1(1.0) | 0 | 1(4.3) | 0 |
| eae EHEC-hly bfpA | 1(1.0) | 1(1.7) | 0 | 0 |
| eae hly shf | 1(1.0) | 1(1.7) | 0 | 0 |
| eae hly irp2 | 1(1.0) | 1(1.7) | 0 | 0 |
| eae hly astA | 1(1.0) | 1(1.7) | 0 | 0 |
| eae astA shf | 1(1.0) | 0 | 0 | 1(5.9) |
| eae shf irp2 | 1(1.0) | 0 | 1(4.3) | 0 |
| eae astA | 1(1.0) | 1(1.7) | 0 | 0 |
| eae cdt | 1(1.0) | 0 | 1(4.3) | 0 |
aEPEC, enteropathogenic Escherichia coli; EHEC, enterohemorrhagic E. coli.
bAll strains hybridized with the locus of enterocyte effacement (LEE) A, LEE B, LEE C, and LEE D probes constructed by McDaniel et al. (12), which suggested that they bear a complete LEE region.
Page created: June 15, 2011
Page updated: June 15, 2011
Page reviewed: June 15, 2011
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.