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Volume 10, Number 3—March 2004

Research

Correlating Epidemiologic Trends with the Genotypes Causing Meningococcal Disease, Maryland

M. Catherine McEllistrem*Comments to Author , John A. Kolano*, Margaret A. Pass†, Dominique A. Caugant‡, Aaron B. Mendelsohn§, Antonio Guilherme Fonseca Pacheco§, Jafar Razeq¶, Lee H. Harrison*†, and the Maryland Emerging Infections Program
Author affiliations: *University of Pittsburgh Graduate School of Public Health and School of Medicine, Pittsburgh, Pennsylvania, USA; †Johns Hopkins University Bloomberg School of Hygiene and Public Health, Baltimore, Maryland, USA; ‡World Health Organization Collaborating Centre for Reference and Research on Meningococci, Norwegian Institute of Public Health, Oslo, Norway; §University of Pittsburgh Graduate School of Public Health, Pittsburgh, Pennsylvania, USA; ¶Maryland Department of Health and Mental Hygiene, Baltimore, Maryland, USA

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Table 1

PFGE and MLST results for selected serogroup C strainsa

Age group (y) Culture date ST No. alleles related to ST-11 ST-11 complex PFGE interpretation
15–24
3/97
11
7/7
Yes
Clonal group 1; 1997 outbreak
15–24
2/97
11
7/7
Yes
Clonal group 1; 1997 outbreak
15–24
7/99
11
7/7
Yes
Clonal group 1; 1999 clone
15–24
5/99
11
7/7
Yes
Clonal group 1; 1999 outbreak
<15
8/99
11
7/7
Yes
Clonal group 1
<15
12/96
11
7/7
Yes
Clonal Group 1
15–24
4/96
11
7/7
Yes
Clonal group 1
<15
6/93
1,626
1/7
No
Nonclonal group 1
<15
5/96
1,623
0/7
No
Nonclonal group 1
<15
6/97
1,060
0/7
No
Nonclonal group 1
>25 6/95 278 0/7 No Nonclonal group 1

aPFGE, pulsed-field gel electrophoresis; ST, sequence typing; MLST, multilocus sequence typing.

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