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Volume 10, Number 3—March 2004

Research

Correlating Epidemiologic Trends with the Genotypes Causing Meningococcal Disease, Maryland

M. Catherine McEllistrem*Comments to Author , John A. Kolano*, Margaret A. Pass†, Dominique A. Caugant‡, Aaron B. Mendelsohn§, Antonio Guilherme Fonseca Pacheco§, Jafar Razeq¶, Lee H. Harrison*†, and the Maryland Emerging Infections Program
Author affiliations: *University of Pittsburgh Graduate School of Public Health and School of Medicine, Pittsburgh, Pennsylvania, USA; †Johns Hopkins University Bloomberg School of Hygiene and Public Health, Baltimore, Maryland, USA; ‡World Health Organization Collaborating Centre for Reference and Research on Meningococci, Norwegian Institute of Public Health, Oslo, Norway; §University of Pittsburgh Graduate School of Public Health, Pittsburgh, Pennsylvania, USA; ¶Maryland Department of Health and Mental Hygiene, Baltimore, Maryland, USA

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Table 2

PFGE and MLST results for selected serogroup Y strains

Age group Culture date ST No. alleles related to ST-23 ST-23 complex PFGE interpretation
15–24
3/93
1,625
6/7
Yes
Clonal group 1
15–24
2/97
1,625
6/7
Yes
Clonal group 1
>25
8/94
1,625
6/7
Yes
Clonal group 1
≥25
7/93
1,625
6/7
Yes
Clonal group 1
<15
8/95
1,622
6/7
Yes
Clonal group 1
<15
2/97
1,622
6/7
Yes
Clonal group 1
>25
3/97
1,622
6/7
Yes
Clonal group 1
<15
5/94
23
7/7
Yes
Clonal group 1
>25
5/96
23
7/7
Yes
Clonal group 1
> 25
6/96
23
7/7
Yes
Clonal group 2
>25
11/99
1,620
5/7
Yes
Clonal group 2
>25
10/99
1,621
6/7
Yes
Clonal group 2
>25
10/99
1,621
6/7
Yes
Clonal group 2
<15 4/99 167 0/7 No Neither clonal group 1or 2

aPFGE, pulsed-field gel electrophoresis; ST, sequence typing; MLST, multilocus sequence typing.

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