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Volume 10, Number 8—August 2004

Research

Antimicrobial Drug Use and Methicillin-resistant Staphylococcus aureus, Aberdeen, 1996–2000

Dominique L. Monnet*, Fiona M. MacKenzie†Comments to Author , José María López-Lozano‡, Arielle Beyaert§, Máximo Camacho§, Rachel Wilson†, David Stuart†, and Ian M. Gould†
Author affiliations: *Statens Serum Institut, Copenhagen, Denmark; †Aberdeen Royal Infirmary, Aberdeen, Scotland; ‡Hospital Vega Baja, Orihuela (Alicante), Spain; §University of Murcia, Murcia, Spain

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Figure 2

Examples of graphic exploration of the relationship between the monthly % methicillin-resistant Staphylococcus aureus (%MRSA) and the monthly use of individual classes of antimicrobials, Aberdeen Royal Infirmary, January 1996–December 2000 (THICK LINE, %MRSA; THIN LINE, Antimicrobial use, 5-month moving average, right Y-axis); A) penicillins with β-lactamase inhibitors, B) macrolides, C) third-generation cephalosporins, D) fluoroquinolones, E) tetracyclines, and F) aminoglycosides.

Figure 2. Examples of graphic exploration of the relationship between the monthly % methicillin-resistant Staphylococcus aureus (%MRSA) and the monthly use of individual classes of antimicrobials, Aberdeen Royal Infirmary, January 1996–December 2000 (THICK LINE, %MRSA; THIN LINE, Antimicrobial use, 5-month moving average, right Y-axis); A) penicillins with β-lactamase inhibitors, B) macrolides, C) third-generation cephalosporins, D) fluoroquinolones, E) tetracyclines, and F) aminoglycosides.

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