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Volume 12, Number 2—February 2006

Letter

Congenital Visceral Leishmaniasis

Catharina C. Boehme*Comments to Author , Ulrike Hain*, Astrid Novosel*, Susanna Eichenlaub*, Erna Fleischmann*, and Thomas Löscher*
Author affiliations: *University of Munich, Munich, Germany

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Figure

A) Immunoblot of the patient (strip no. 21) and the patient's mother (strip no. 22) showing specific antibodies against 14- and 16-kDa proteins of Leishmania infantum. B) restriction fragment length polymorphism patterns after HaeIII digestion of the ribosomal internal transcribed spacer 1 polymerase chain reaction products. P, patient; Li, L. infantum; Lm, L. major; Lt, L. tropica; M, 100-bp ladder.

Figure. A) Immunoblot of the patient (strip no. 21) and the patient's mother (strip no. 22) showing specific antibodies against 14- and 16-kDa proteins of Leishmania infantum. B) restriction fragment length polymorphism patterns after HaeIII digestion of the ribosomal internal transcribed spacer 1 polymerase chain reaction products. P, patient; Li, L. infantum; Lm, L. major; Lt, L. tropica; M, 100-bp ladder.

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