Afia Zafar* , Robert Swanepoel†, Roger Hewson‡, Mazhar Nizam§, Altaf Ahmed§, Akhtar Husain*, Antoinette Grobbelaar†, Kevin Bewley‡, Valerie Mioulet‡, Barry Dowsett‡, Linda Easterbrook‡, and Rumina Hasan*
Author affiliations: *Aga Khan University Hospital, Karachi, Pakistan; †National Institute for Communicable Diseases, Sandringham, South Africa; ‡Centre for Emergency Preparedness and Response, Porton Down, Salisbury, UK; §Patel Hospital, Karachi, Pakistan;
Figure 1. Nosocomial buffalopoxvirus infection of patients in burns units. A) Lesions involving intact skin around a burn wound and the wound itself. B) Lesions around an insertion site for an intravenous line. C) Orthopoxvirus particles detected by electron microscopy (EM) examination of negatively stained grids prepared from pustular material (magnification ×73,000). D) Transmission EM examination of ultrathin sections of infected Vero cell cultures showing classic intracytoplasmic orthopoxvirus factories and maturing virus particles (magnification ×21,000).
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