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Volume 15, Number 9—September 2009

Research

Genetics and Pathogenesis of Feline Infectious Peritonitis Virus

Meredith A. BrownComments to Author , Jennifer L. Troyer, Jill Pecon-Slattery, Melody E. Roelke, and Stephen J. O’Brien
Author affiliations: National Cancer Institute, Frederick, Maryland, USA (M.A. Brown, J. Pecon-Slattery, S.J. O’Brien); SAIC-Frederick, Inc., Frederick (J.L. Troyer, M.E. Roelke)

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Figure 2

A) Histopathologic and immunohistochemical (IHC) results from 23 necropsied cats positive for antibodies against feline coronavirus. Liver, lung, spleen, colon, jejunum, stomach, heart, kidney, lymph node were evaluated by IHC. Feline infectious peritonitis (FIP) cases are highlighted in gray. Pos, positive; Neg, negative; ND, not done. B) Representative tissues from cat no. FCA-4653, spleen (histopathologic) showing granuloma (arrow); magnification ×20. C) Representative tissues from cat no. FC

Figure 2. A) Histopathologic and immunohistochemical (IHC) results from 23 necropsied cats positive for antibodies against feline coronavirus. Liver, lung, spleen, colon, jejunum, stomach, heart, kidney, lymph node were evaluated by IHC. Feline infectious peritonitis (FIP) cases are highlighted in gray. Pos, positive; Neg, negative; ND, not done. B) Representative tissues from cat no. FCA-4653, spleen (histopathologic) showing granuloma (arrow); magnification ×20. C) Representative tissues from cat no. FCA-4590, small intestine (IHC); magnification ×20. D) Red staining indicates binding of coronavirus antibody (CoV p56, arrow); magnification ×100.

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