Emerging Infectious Disease ISSN: 1080-6059

Dual and Recombinant Infections: An Integral Part of the HIV-1 Epidemic in Brazil

Artur Ramos*†, Amilcar Tanuri†, Mauro Schechter†, Mark A. Rayfield*, Dale J. Hu*, Maulori C. Cabral†, Claudiu I. Bandea*, James Baggs‡, and Danuta Pieniazek*

Author affiliations: *Centers for Disease Control and Prevention, Atlanta, Georgia, USA; †Universidade Federal do Rio de Janeiro, Brazil; ‡Emory University, Atlanta, Georgia, USA

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Figure 1

Differentiation between single (A) and dual (B) HIV-1 infections by the restriction fragment length polymorphism analysis of the polymerase chain reaction—amplified prt. A: Three AluI digestion patterns represent subtypes A, C, and F (pattern 1) and subtypes B and D (patterns 2 and 3); two HinfI patterns represent subtypes D (pattern 1) and B (pattern 2); two BclI patterns represent subtypes F (patterns 1) and A and C (pattern 2); two ScaI patterns represent subtypes A (pattern 1) and C (pattern

Figure 1. Differentiation between single (A) and dual (B) HIV-1 infections by the restriction fragment length polymorphism analysis of the polymerase chain reaction—amplified prt. A: Three AluI digestion patterns represent subtypes A, C, and F (pattern 1) and subtypes B and D (patterns 2 and 3); two HinfI patterns represent subtypes D (pattern 1) and B (pattern 2); two BclI patterns represent subtypes F (patterns 1) and A and C (pattern 2); two ScaI patterns represent subtypes A (pattern 1) and C (pattern 2). B: Two AluI digestion patterns (1 and 2) in the dually infected patient with HIV-1 subtypes F and B; two HinfI patterns (1 and 2) in the patient infected with subtypes B and D viruses.

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