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Volume 5, Number 1—February 1999

Research

Dual and Recombinant Infections: An Integral Part of the HIV-1 Epidemic in Brazil

Artur Ramos*†, Amilcar Tanuri†, Mauro Schechter†, Mark A. Rayfield*, Dale J. Hu*, Maulori C. Cabral†, Claudiu I. Bandea*, James Baggs‡, and Danuta Pieniazek*Comments to Author 
Author affiliations: *Centers for Disease Control and Prevention, Atlanta, Georgia, USA; †Universidade Federal do Rio de Janeiro, Brazil; ‡Emory University, Atlanta, Georgia, USA

Main Article

Table 2

p17/p24 gag, prt, and C2-V3 genetic subtyping of HIV-1 DNA sequencesa from peripheral blood mononuclear cells collected from 18 patients in Rio de Janeiro

Genotypes
Specimen No. gag p17 gag p24 prt C2-V3
Group 1b 8 NDc B B B
Group 2d 4 F F F F
(BR46) (NAe) (B) (F) (F)
(BR59) (F) (B) (F) (F)
(BR57) (F) (B/F) (F) (F)
(BR97) (B) (B/F) (F) (F)
(BR60) (B) (B/F) (F) (B)
(BR43) (B) (B) (F) (B)

aRecombinant specimens are shown in parentheses; B/F mosaic structure within a 311-bp of the p24 gag fragment consisting of subtypes B and F sequences.
bGroup 1: BR34, BR52, BR55, BR64, BR65, BR71, BR75, and BR92.
cND=not done.
dGroup 2: BR41, BR54, BR58, and BR112.
eNA = not available due to negative PCR.

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