Volume 12, Number 12—December 2006
Atypical BSE is probably not sporadic and not related to sporadic Creutzfeldt-Jakob disease.
This technique can be used to study the geography and ecology of disease transmission.
The probability of further human infection is low and the risk is further mitigated by rodent import restrictions.
Risk factors include direct or household contact with sick or dead poultry and absence of an indoor water source.
Greater sampling and monitoring efforts are needed to reverse a significant increase in prevalence.
Human metapneumovirus causes clinical signs in turkey poults.
Infection with Leishmania tropica is emerging because of encroachment of rock hyraxes and transmission by multiple vector species.
PCR and sequencing provide the first evidence that B. quintana can be pathogenic in dogs.
A serosurvey showed that this virus is widespread in dogs.
Campylobacter, Mycoplasma pneumoniae, and influenza (or influenza vaccination) act as infectious triggers for Guillain-Barré syndrome.
Nipah virus was likely transmitted from fruit bats to humans by drinking fresh date palm sap.
The green lizard is implicated in the transmission cycle of B. lusitaniae.
We identified 4 discrete Plasmodium spp. sequences from the blood of orangutans, including 1 of P. vivax, which has implications for human residents and orangutan rehabilitation programs.
The primary agent of neurotropic Lyme disease in Eurasia was found in seabird ticks in northeastern North America.
Lagos bat virus from water mongoose showed strong sequence homology with other Lagos bat virus isolates from South Africa.
Low-intensity cattle grazing limits Borrelia burgdorferi s.l., but not B. miyamotoi, in vector ticks.
Healthcare workers in hospitals affected by SARS experience increased psychological stress 1–2 years after the outbreak.
Prevalence of colonization was 6.5%, and employment within a large-animal practice was a significant risk factor.
Livestock may serve as an important source of community-acquired MRSA in a low-prevalence situation.
When syndromic surveillance detected a substantial proportion of outbreaks before clinical case finding, false-positive results occurred.
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