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Geographically Distinct Circulation of Genotype II and III St. Louis Encephalitis Virus, Texas, USA, 2009–2024
Alexander R. Kneubehl, Daniel P. Rehm, Michael W. Curtis, Bianca M. Wimmer, Bethany Bolling, Angie Broussard, Jeremy Vela, Jennifer Rocha, Lindsey Templeton, Maximea Vigilant, Courtney Standlee, Steven M. Presley, Job E. Lopez, and Shannon E. Ronca
Author affiliation: Baylor College of Medicine, Houston, Texas, USA (A.R. Kneubehl, D.P. Rehm, M.W. Curtis, J.E. Lopez, S.E. Ronca); Texas Children’s Hospital William T. Schearer Center for Human Immunobiology, Houston (A.R. Kneubehl, D.P. Rehm, S.E. Ronca); Texas Tech University Institute of Environmental and Human Health, Lubbock, Texas, USA (B.M. Wimmer, S.M. Presley); Texas Department of State Health Services, Austin, Texas, USA (B. Bolling); Harris County Public Health Division of Mosquito and Vector Control, Houston (A. Broussard, J. Vela, J. Rocha, L. Templeton, M. Vigilant, C. Standlee)
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Figure 3
Figure 3. Maximum-likelihood phylogenic analysis of all St. Louis encephalitis virus genomes from study of circulation of genotype II and III St. Louis encephalitis virus, Texas, USA, 2009–2024. Maximum-likelihood inferred tree shows 276 genomes currently available that have <30% of the genome missing. Tip color represents sample’s country of origin; samples from Texas are labeled with a separate color (red) to distinguish Texas samples from rest of United States. Genotypes annotated on the tree indicate which clade contains which genotype or genotypes. Tree was midpoint rooted. Scale bar indicates number of nucleotide substitutions per site.
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