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Volume 18, Number 2—February 2012

Dispatch

Shuni Virus as Cause of Neurologic Disease in Horses

Charmaine van Eeden, June H. Williams, Truuske G.H. Gerdes, Erna van Wilpe, Adrianne Viljoen, Robert Swanepoel, and Marietjie VenterComments to Author 
Author affiliations: University of Pretoria, Pretoria, South Africa (C. van Eeden, J.H. Williams, E. van Wilpe, A. Viljoen, R. Swanepoel); Onderstepoort Veterinary Institute, Pretoria (T.G.H. Gerdes); National Institute for Communicable Diseases, Sandringham, South Africa (M. Venter)

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Figure 2

Maximum-likelihood tree constructed under the HKY codon position substitution model using PhyML (http://code.google.com/p/phyml/) of a 330-bp fragment of small segment RNA of Shuni virus (SHUV) identified in horses in South Africa, with representative sequences of selected other orthobunyaviruses. Scale bar = 0.07 nt substitutions. Estimates were based on bootstrap resampling conducted with 100 replicates. Only values >70 are shown. All SHUV amplicons were sequenced, and the data were deposit

Figure 2. Maximum-likelihood tree constructed under the HKY codon position substitution model using PhyML (http://code.google.com/p/phyml/) of a 330-bp fragment of small segment RNA of Shuni virus (SHUV) identified in horses in South Africa, with representative sequences of selected other orthobunyaviruses. Scale bar = 0.07 nt substitutions. Estimates were based on bootstrap resampling conducted with 100 replicates. Only values >70 are shown. All SHUV amplicons were sequenced, and the data were deposited in GenBank, accession nos. SAE 18/09–HQ610137, SAE 72/09–HQ610138, SAE 27/10–HQ 610139, SAE 38/10–HQ 610140, SAE 39/10–HQ 610141, SAE 48/10–HQ 610142, and SAE 109/10–HQ 610143. Reference strains and GenBank accession numbers are indicated.

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