Shuni Virus as Cause of Neurologic Disease in Horses
Charmaine van Eeden, June H. Williams, Truuske G.H. Gerdes, Erna van Wilpe, Adrianne Viljoen, Robert Swanepoel, and Marietjie Venter
Author affiliations: University of Pretoria, Pretoria, South Africa (C. van Eeden, J.H. Williams, E. van Wilpe, A. Viljoen, R. Swanepoel); Onderstepoort Veterinary Institute, Pretoria (T.G.H. Gerdes); National Institute for Communicable Diseases, Sandringham, South Africa (M. Venter)
Figure 2. Maximum-likelihood tree constructed under the HKY codon position substitution model using PhyML (http://code.google.com/p/phyml/) of a 330-bp fragment of small segment RNA of Shuni virus (SHUV) identified in horses in South Africa, with representative sequences of selected other orthobunyaviruses. Scale bar = 0.07 nt substitutions. Estimates were based on bootstrap resampling conducted with 100 replicates. Only values >70 are shown. All SHUV amplicons were sequenced, and the data were deposited in GenBank, accession nos. SAE 18/09–HQ610137, SAE 72/09–HQ610138, SAE 27/10–HQ 610139, SAE 38/10–HQ 610140, SAE 39/10–HQ 610141, SAE 48/10–HQ 610142, and SAE 109/10–HQ 610143. Reference strains and GenBank accession numbers are indicated.
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