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Volume 11, Number 1—January 2005

Research

Genetic Background of Escherichia coli and Extended-spectrum β-Lactamase Type

Author affiliations: *Hôpital Louis Mourier, AP-HP, Colombes, France; †INSERM E0339, Paris, France; ‡Hôpital Nord, Amiens, France; §Hôpital Tenon, Paris, France; ¶Hôpital Trousseau, Paris, France; #Hôpital Morvan, Brest, France

Main Article

Figure

Graphic representation of the results of the factorial analysis of correspondence carried out with whole data from the 129 Escherichia coli strains. A) Projections of the variables on the F1/F2 plane: phylogenetic group and subgroups (A0, A1, B1, B22, B23, D1, and D2), type of extended-spectrum β-lactamase (ESBL) (TEM, SHV, CTX-M), virulence factors (pap, sfa, hly, aer), and the source infection (inf) or colonization (col). B) Projections of the variables on the F1/F2 plane: phylogenetic group a

Figure. Graphic representation of the results of the factorial analysis of correspondence carried out with whole data from the 129 Escherichia coli strains. A) Projections of the variables on the F1/F2 plane: phylogenetic group and subgroups (A0, A1, B1, B22, B23, D1, and D2), type of extended-spectrum β-lactamase (ESBL) (TEM, SHV, CTX-M), virulence factors (pap, sfa, hly, aer), and the source infection (inf) or colonization (col). B) Projections of the variables on the F1/F2 plane: phylogenetic group and subgroups (A0, A1, B1, B22, B23, D1, and D2), type of ESBL (TEM, SHV, CTX-M), and ciprofloxacin resistance (cipR) or the ciprofloxacin susceptibility (cipS).

Main Article

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