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Volume 13, Number 12—December 2007

Research

Phenotypic Similarity of Transmissible Mink Encephalopathy in Cattle and L-type Bovine Spongiform Encephalopathy in a Mouse Model

Thierry G.M. Baron*Comments to Author , Anna Bencsik*, Anne-Gaëlle Biacabe*, Eric Morignat*, and Richard A. Bessen†
Author affiliations: *Agence Française de Sécurité Sanitaire des Aliments–Lyon, Lyon, France; †Montana State University, Bozeman, Montana, USA;

Main Article

Figure 1

Western blot analyses of protease-resistant prion protein from proteinase K–treated brain homogenates from cattle transmissible spongiform encephalopathies (TSEs). Typical bovine spongiform encephalopathy (BSE) (lanes 1, 5), L-type BSE (lane 2), transmissible mink encephalopathy (TME) in cattle (lane 3), H-type BSE (lane 4). Bars to the left of the panel indicate the 29.0- and 20.1-kDa marker positions.

Figure 1. Western blot analyses of protease-resistant prion protein from proteinase K–treated brain homogenates from cattle transmissible spongiform encephalopathies (TSEs). Typical bovine spongiform encephalopathy (BSE) (lanes 1, 5), L-type BSE (lane 2), transmissible mink encephalopathy (TME) in cattle (lane 3), H-type BSE (lane 4). Bars to the left of the panel indicate the 29.0- and 20.1-kDa marker positions.

Main Article

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