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Volume 22, Number 10—October 2016
Synopsis

Population-Level Effects of Human Papillomavirus Vaccination Programs on Infections with Nonvaccine Genotypes

David MesherComments to Author , Kate Soldan, Matti Lehtinen, Simon Beddows, Marc Brisson, Julia M.L. Brotherton, Eric P.F. Chow, Teresa Cummings, Mélanie Drolet, Christopher K. Fairley, Suzanne M. Garland, Jessica A. Kahn, Kimberley Kavanagh, Lauri E. Markowitz, Kevin G. Pollock, Anna Söderlund-Strand, Pam Sonnenberg, Sepehr N. Tabrizi, Clare Tanton, Elizabeth R. Unger, and Sara L. Thomas
Author affiliations: Public Health England, London, UK (D. Mesher, K. Soldan, S. Beddows); London School of Hygiene and Tropical Medicine, London (D. Mesher, S.L. Thomas); University of Tampere, Tampere, Finland (M. Lehtinen); Imperial College London, London (M. Brisson); Centre de recherche du CHU de Québec, Quebec City, Quebec, Canada (M. Brisson, M. Drolet); Université Laval, Quebec (M. Brisson, M Drolet); Victorian Cytology Service, Melbourne, Victoria, Australia (J.M.L. Brotherton); University of Melbourne, Melbourne (J.M.L. Brotherton, S.M. Garland, S.N. Tabrizi); Melbourne Sexual Health Centre, Melbourne (E.P.F. Chow, C.K. Fairley); Monash University, Melbourne (E.P.F. Chow, C.K. Fairley); Indiana University School of Medicine, Indianapolis, Indiana, USA (T. Cummings); Murdoch Childrens Research Institute, Parkville, Victoria, Australia (S.M. Garland, S.N. Tabrizi); The Royal Women’s Hospital, Melbourne (S.M. Garland, S.N. Tabrizi); Cincinnati Children’s Hospital Medical Center, Cincinnati, Ohio, USA (J.A. Kahn); University of Cincinnati College of Medicine, Cincinnati (J.A. Kahn); University of Strathclyde, Glasgow, Scotland, UK (K. Kavanagh); Centers for Disease Control and Prevention, Atlanta, Georgia, USA (L. Markowitz, E. Unger); Health Protection Scotland, Glasgow (K.G. Pollock); Division of Clinical Microbiology, Laboratory Medicine, Skåne, Lund, Sweden (A. Söderlund-Strand); University College London, London (P. Sonnenberg, C. Tanton)

Main Article

Figure 2

Prevalence ratios and 95% CIs for high-risk human papillomavirus (HPV) types (HPV31, HPV33, and HPV45) that had evidence of cross-protection for girls and women <19 years of age and women 20–24 years of age in studies included in a meta-analysis of changes in prevalences of nonvaccine HPV genotypes after introduction of HPV vaccination. A) HPV31; B) HPV33; C) HPV45. Percentages in brackets represent vaccination coverage (>1 dose) for each study and age group. The size of the gray boxes aro

Figure 2. Prevalence ratios and 95% CIs for high-risk human papillomavirus (HPV) types (HPV31, HPV33, and HPV45) that had evidence of cross-protection for girls and women <19 years of age and women 20–24 years of age in studies included in a meta-analysis of changes in prevalences of nonvaccine HPV genotypes after introduction of HPV vaccination. A) HPV31; B) HPV33; C) HPV45. Percentages in brackets represent vaccination coverage (>1 dose) for each study and age group. The size of the gray boxes around the plot points indicates the relative weight given to each study in the calculation of the summary estimate. The study by Cameron et al. (25) is omitted from analyses for the younger age group because this study included no data for the group <19 years of age. The study by Cummings et al. (21) is omitted from analyses for women 20–24 years of age because this study included no data for this age group. Pre, prevaccination; post, postvaccination.

Main Article

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Page updated: September 20, 2016
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The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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