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Volume 13, Number 11—November 2007
Research

Non-A Hepatitis B Virus Genotypes in Antenatal Clinics, United Kingdom

Samir Dervisevic*Comments to Author , Samreen Ijaz†, Shahneila Chaudry*, and Richard S. Tedder*†
Author affiliations: *University College London Hospitals National Health Service Foundation Trust, London, United Kingdom; †Health Protection Agency, London, United Kingdom;

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Figure

Box and whisker plots of hepatitis B virus (HBV) load in 3 groups of mothers whose serum contained hepatitis B virus e antigen (HBeAg), antibody to hepatitis B virus e antigen (anti-HBe), or neither of these markers (e Neg). Boxes are middle quartiles, horizontal lines are medians, whiskers are ranges, and dots represent 10 anti-HBe–seropositive mothers whose serum contained >104 IU/mL HBV DNA. Thirty-three anti-HBe–seropositive mothers and 1 mother whose serum did not contain either marker did not have detectable HBV DNA (<50 IU/mL).

Figure. Box and whisker plots of hepatitis B virus (HBV) load in 3 groups of mothers whose serum contained hepatitis B virus e antigen (HBeAg), antibody to hepatitis B virus e antigen (anti-HBe), or neither of these markers (e Neg). Boxes are middle quartiles, horizontal lines are medians, whiskers are ranges, and dots represent 10 anti-HBe–seropositive mothers whose serum contained >104 IU/mL HBV DNA. Thirty-three anti-HBe–seropositive mothers and 1 mother whose serum did not contain either marker did not have detectable HBV DNA (<50 IU/mL).

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