Volume 12, Number 3—March 2006
Research
Pneumonic Plague Cluster, Uganda, 2004
Elizabeth M. Begier*
, Gershim Asiki†, Zaccheus Anywaine†, Brook Yockey‡, Martin Schriefer‡, Philliam Aleti§, Asaph Ogen-Odoi§, J. Erin Staples*‡, Christopher Sexton‡, Scott Bearden‡, and Jacob L. Kool‡
, Gershim Asiki†, Zaccheus Anywaine†, Brook Yockey‡, Martin Schriefer‡, Philliam Aleti§, Asaph Ogen-Odoi§, J. Erin Staples*‡, Christopher Sexton‡, Scott Bearden‡, and Jacob L. Kool‡
Figure 1
Figure 1. A) Grossly bloody sputum sample obtained from the surviving patient (caregiver B2) 30 h after onset of primary pneumonic plague. B) Polymerase chain reaction results of sputum sample from caregiver B2. Lanes 1–3, caf1; lanes 4–6, repA1; lanes 7–9, pla. Lanes 1, 4, and 7 are positive controls; lanes 2, 5, and 8 are patient samples; lanes 3, 6, and 9 are negative controls. C) Anti-F1 direct fluorescent antibody staining of sputum sample from caregiver B2. Numerous bacteria with classic halo structures are characteristic of Yersinia pestis. The circled bacterium classically depicts this halo.
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