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Volume 14, Number 3—March 2008
Research

Genetic Variability of West Nile Virus in US Blood Donors, 2002–2005

Andriyan Grinev*, Sylvester Daniel*, Susan L. Stramer†, Susan N. Rossmann‡, Sally Caglioti§, and Maria Rios*Comments to Author 
Author affiliations: *Food and Drug Administration, Bethesda, Maryland, USA; †American Red Cross, Gaithersburg, Maryland, USA; ‡Gulf Coast Regional Blood Center, Houston, Texas, USA; §Blood System Laboratories, Tempe, Arizona, USA;

Main Article

Figure 2

Distance analysis of envelope glycoprotein of West Nile virus isolates collected during 1999–2006 epidemics in the United States. Phylogram is based on maximum parsimony analysis of complete nucleotide sequences of the envelope gene. Diamonds indicate isolates from this study. All isolates from clade 2 (WN02 strain) contained conserved mutations at positions 1442 (T → C) and 2466 (C → T). Values near branches represent percentage support by parsimony bootstrap analysis. Scale bar represents a 1-nt change.

Figure 2. Distance analysis of envelope glycoprotein of West Nile virus isolates collected during 1999–2006 epidemics in the United States. Phylogram is based on maximum parsimony analysis of complete nucleotide sequences of the envelope gene. Diamonds indicate isolates from this study. All isolates from clade 2 (WN02 strain) contained conserved mutations at positions 1442 (T → C) and 2466 (C → T). Values near branches represent percentage support by parsimony bootstrap analysis. Scale bar represents a 1-nt change.

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Page created: July 07, 2010
Page updated: July 07, 2010
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The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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