Fosfomycin Resistance in Escherichia coli, Pennsylvania, USA
Hind Alrowais, Christi L. McElheny, Caressa N. Spychala, Sangeeta Sastry, Qinglan Guo, Adeel A. Butt, and Yohei Doi
Author affiliations: University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, USA (H. Alrowais, C.L. McElheny, C.N. Spychala, S. Sastry, Q. Guo, A.A. Butt, Y. Doi); Fudan University, Shanghai, China (Q. Guo); Ministry of Health, Shanghai (Q. Guo); Hamad Healthcare Quality Institute, Doha, Qatar (A.A. Butt); Hamad Medical Corporation, Doha (A.A. Butt)
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Figure 1
Figure 1. Comparative analysis of fosA3-carrying IncFII plasmids pYHCC with pXZ. Open reading frames are indicated by arrows and colored according to their putative functions: magenta arrows indicate genes involved in replication; blue arrows indicate genes associated with plasmid conjugal transfer. Brown arrows indicate genes involved in plasmid stability; red arrows indicate antimicrobial drug resistance genes; green arrows indicate accessory genes of mobile elements; gray arrows indicate other backbone genes and inserted foreign genes. Light blue shading indicates shared backbone regions with a high degree of homology. MRR, multidrug resistance region.
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