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Volume 22, Number 7—July 2016

Dispatch

Clinical Manifestations of Senecavirus A Infection in Neonatal Pigs, Brazil, 2015

Raquel A. Leme1, Thalita E.S. Oliveira1, Brígida K. Alcântara, Selwyn A. Headley, Alice F. Alfieri, Ming Yang, and Amauri A. AlfieriComments to Author 
Author affiliations: Universidade Estadual de Londrina, Paraná, Brazil (R.A. Leme, T.E.S. Oliveira, B.K. Alcântara, S.A. Headley, A.F. Alfieri, A.A. Alfieri); National Centre for Foreign Animal Disease, Winnipeg, Manitoba, Canada (M. Yang)

Main Article

Figure 2

Phylogenetic relationship of strains of Senecavirus A identified in Brazil during 2015 (black circles) and other sequences available in GenBank derived from species of picornavirus associated with vesicular disease. Maximum-likelihood phylogenetic tree construction used the Kimura 2-parameter model with γ distribution based on the partial viral protein (VP) 3/VP1 region of the Senecavirus A genome. GenBank accession numbers are given in parentheses. Bootstrap values determined in 1,000 replicati

Figure 2. Phylogenetic relationship of strains of Senecavirus A identified in Brazil during 2015 (black circles) and other sequences available in GenBank derived from species of picornavirus associated with vesicular disease. Maximum-likelihood phylogenetic tree construction used the Kimura 2-parameter model with γ distribution based on the partial viral protein (VP) 3/VP1 region of the Senecavirus A genome. GenBank accession numbers are given in parentheses. Bootstrap values determined in 1,000 replication. Scale bar indicates nucleotide substitutions per site.

Main Article

1These authors contributed equally to this article.

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