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Volume 23, Number 8—August 2017

Synopsis

Characteristics of Dysphagia in Infants with Microcephaly Caused by Congenital Zika Virus Infection, Brazil, 2015

Mariana C. LealComments to Author , Vanessa van der Linden, Thiago P. Bezerra, Luciana de Valois, Adriana C.G. Borges, Margarida M.C. Antunes, Kátia G. Brandt, Catharina X. Moura, Laura C. Rodrigues, and Coeli R. Ximenes
Author affiliations: Hospital Agamenon Magalhães, Recife, Brazil (M.C. Leal); Universidade Federal de Pernambuco, Recife (M.C. Leal, T.P. Bezerra, M.M.C. Antunes, K.G. Brandt, C.R. Ximenes); Hospital Barão de Lucena, Recife (V. van der Linden); Association for Assistance of Disabled Children, Recife (V. van der Linden, L. de Valois); Prived Clinic, Recife (A.C.G. Borges); Real Hospital Português, Recife (C.X. Moura); London School of Hygiene and Tropical Medicine, London, UK (L.C. Rodrigues)

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Figure 1

Computed tomography radiographs of the brains of 2 infants with dysphagia and microcephaly caused by congenital Zika virus infection, Brazil, 2015. A–D) Images for patient 4 show malformation of cortical development, ventriculomegaly (long arrows), and calcifications in cortical and subcortical white matter in transition between cortex and white matter (short arrows). E–H) Images for patient 6 show no malformation of cortical development or ventriculomegaly, but calcifications are visible in the

Figure 1. Computed tomography radiographs of the brains of 2 infants with dysphagia and microcephaly caused by congenital Zika virus infection, Brazil, 2015. A–D) Images for patient 4 show malformation of cortical development, ventriculomegaly (long arrows), and calcifications in cortical and subcortical white matter in transition between cortex and white matter (short arrows). E–H) Images for patient 6 show no malformation of cortical development or ventriculomegaly, but calcifications are visible in the cortical area (arrows).

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