Phylogeny and Immunoreactivity of Norovirus GII.P16-GII.2, Japan, Winter 2016–17
Koo Nagasawa
1, Yuki Matsushima
1, Takumi Motoya, Fuminori Mizukoshi, Yo Ueki, Naomi Sakon, Koichi Murakami, Tomomi Shimizu, Nobuhiko Okabe, Noriko Nagata, Komei Shirabe, Hiroto Shinomiya, Wataru Suzuki, Makoto Kuroda, Tsuyoshi Sekizuka, Akihide Ryo, Kiyotaka Fujita, Kazunori Oishi, Kazuhiko Katayama
, and Hirokazu Kimura
Author affiliations: National Institute of Infectious Diseases,Tokyo, Japan (K. Nagasawa, K. Murakami, M. Kuroda, T. Sekizuka, K. Oishi, H. Kimura); Kawasaki City Institute for Public Health, Kanagawa, Japan (Y. Matsushima, T. Shimizu, N. Okabe); Ibaraki Prefectural Institute of Public Health, Ibaraki, Japan (T. Motoya, N. Nagata); Tochigi Prefectural Institute of Public Health and Environmental Science, Tochigi, Japan (F. Mizukoshi); Miyagi Prefectural Institute of Public Health and Environment, Miyagi, Japan (Y. Ueki); Osaka Prefectural Institute of Public Health, Osaka, Japan (N. Sakon); Yamaguchi Prefectural Institute of Public Health and Environment, Yamaguchi, Japan (K. Shirabe); Ehime Prefectural Institute of Public Health and Environmental Science, Ehime, Japan (H. Shinomiya); Eiken Chemical Co. Ltd., Tochigi (W. Suzuki); Yokohama City University Graduate School of Medicine, Kanagawa (A. Ryo, H. Kimura); Gumma Paz University, Gunma, Japan (K. Fujita); Kitasato University, Minato-ku, Tokyo (K. Katayama)
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Figure
Figure. Phylogenetic trees for the A) capsid (VP1) gene and B) RNA-dependent RNA polymerase (RdRp) region in human norovirus GII strains. Trees were constructed by using the maximum-likelihood method. Bold letters denote GII.2v strains. Numbers at branch nodes show bootstrap values with >70% support. Scale bar represents number of nucleotide substitutions per site.
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