Isolation of Diverse Simian Arteriviruses Causing Hemorrhagic Disease
Teressa M. Shaw, Samuel T. Dettle, Andres Mejia, Jennifer M. Hayes, Heather A. Simmons, Puja Basu, Jens H. Kuhn, Mitchell D. Ramuta, Cody J. Warren, Peter B. Jahrling, David H. O’Connor, Liupei Huang, Misbah Zaeem, Jiwon Seo, Igor I. Slukvin, Matthew E. Brown, and Adam L. Bailey
Author affiliations: University of Wisconsin, Madison, Wisconsin, USA (T.M. Shaw, S.T. Dettle, A. Mejia, M.D. Ramuta, D.H. O’Connor, L. Huang, M. Zaeem, J. Seo, I.I. Slukvin, M.E. Brown, A.L. Bailey); Wisconsin National Primate Research Center, Madison (S.T. Dettle, A. Mejia, J.M. Hayes, H.A. Simmons, P. Basu, D.H. O’Connor, I.I. Slukvin); National Institutes of Health, Fort Detrick, Frederick, Maryland, USA (J.H. Kuhn, P.B. Jahrling); The Ohio State University, Columbus, Ohio, USA (C.J. Warren)
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Figure 7
Figure 7. Cytopathic effect after infection of induced pluripotent stem cell–derived macrophages in study of diverse simarteriviruses causing hemorrhagic disease. Induced pluripotent stem cells from crab-eating macaques were differentiated into macrophages and mock infected (uninfected) or infected with different simarteriviruses. Cytopathic effect was monitored for 0–3 dpi. Original magnification ×100. dpi, days after inoculation; KRCV-1, Kibale red colobus monkey virus 1; PBJV, Pebjah virus; SHFV, simian hemorrhagic fever virus; SWBV-1, Southwest baboon virus 1.
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