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Volume 1, Number 3—July 1995
Synopsis

Streptococcal Toxic-Shock Syndrome: Spectrum of Disease, Pathogenesis, and New Concepts in Treatment

Dennis L. Stevens
Author affiliation: Professor of Medicine, University of Washington School of Medicine, Seattle,Washington
Chief, Infectious Disease Section, Veterans Affairs Medical Center, Boise, Idaho, USA

Main Article

Figure

Pathogenesis of scarlet fever, bacteremia, and toxic shock syndrome. M-1+SPEA+=aGAS strain that contains M protein type 1 and streptococcal pyrogenic exotoxin A (SPEA); + anti-M-1 = the presence of antibody to M protein type 1; -anti-M-1 = the absence of antibody to M protein type 1;anti-SPEA+=antibody to SPEA; and DIC = disseminated intravascular coagulation.

Figure. Pathogenesis of scarlet fever, bacteremia, and toxic shock syndrome. M-1+SPEA+=aGAS strain that contains M protein type 1 and streptococcal pyrogenic exotoxin A (SPEA); + anti-M-1 = the presence of antibody to M protein type 1; -anti-M-1 = the absence of antibody to M protein type 1;anti-SPEA+=antibody to SPEA; and DIC = disseminated intravascular coagulation.

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