Shiga Toxin–producing Escherichia coli Strains Negative for Locus of Enterocyte Effacement
Hayley J. Newton
1, Joan Sloan
1, Dieter M. Bulach, Torsten Seemann, Cody C. Allison, Marija Tauschek, Roy M. Robins-Browne, James C. Paton, Thomas S. Whittam, Adrienne W. Paton, and Elizabeth L. Hartland
Author affiliations: Monash University, Melbourne, Victoria, Australia (H.J. Newton, J. Sloan, D.M. Bulach, T. Seemann, C.C. Allison, E.L. Hartland); University of Melbourne, Melbourne (H.J. Newton, J. Sloan, M. Tauschek, R.M. Robins-Browne, E.L. Hartland); University of Adelaide, Adelaide, South Australia, Australia (J.C. Paton, A.W. Paton); Michigan State University, East Lansing, Michigan, USA (T.S. Whittam)
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Figure 3
Figure 3. Neighbor-joining tree of ehxA (A) and repA (B) as implemented in ClustalW (www.ebi.ac.uk/Tools/clustalw2). This rectangular cladogram demonstrates the distinct clades (shown by boxes) for ehxA that delineate locus of enterocyte effacement (LEE)–negative and LEE-positive Shiga toxin–producing Escherichia coli strains. Exceptions to this pattern are shown in boldface, strain names are shown in parentheses, and + or – indicates the presence or absence of LEE. Significant nodes were identified by bootstrapping (Monte Carlo randomization); nodes were present in >70% of the 1,000 bootstrap trees highlighted and identified as significant.
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