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Volume 15, Number 9—September 2009
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Phylogeny and Disease Association of Shiga Toxin–producing Escherichia coli O91

Alexander MellmannComments to Author , Angelika Fruth, Alexander W. Friedrich, Lothar H. Wieler, Dag Harmsen, Dirk Werber, Barbara Middendorf, Martina Bielaszewska, and Helge Karch
Author affiliations: Institute of Hygiene and the National Consulting Laboratory on Hemolytic Uremic Syndrome, Münster, Germany (A. Mellman, A.W. Friedrich, D. Harmsen, B. Middendorf, M. Bielaszewska, H. Karch); Department of Periodontolgy, Münster (D. Harmsen); Robert Koch Institute, Wernigerode, Germany (A. Fruth); Free University Berlin, Berlin, Germany (L.H. Wieler); Robert Koch Institute, Berlin (D. Weber)

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Figure

Minimum spanning tree based on the multilocus sequence typing allelic profiles portraying the clonal distribution of the 100 Escherichia coli O91:H8/H10/H14/H21/H–/Hnt isolates (highlighted in gray) associated with different diseases in relation to the hemolytic uremic syndrome–associated enterohemorrhagic E. coli collection. Each dot represents a given sequence type, and the size of each circle is proportional to the number of strains analyzed. Connecting lines show the number of identical alle

Figure. Minimum spanning tree based on the multilocus sequence typing allelic profiles portraying the clonal distribution of the 100 Escherichia coli O91:H8/H10/H14/H21/H–/Hnt isolates (highlighted in gray) associated with different diseases in relation to the hemolytic uremic syndrome–associated enterohemorrhagic E. coli collection. Each dot represents a given sequence type, and the size of each circle is proportional to the number of strains analyzed. Connecting lines show the number of identical alleles between 2 STs (thick black line, 6 of 7 alleles identical; thick gray line, 5 alleles identical; thick dashed line, 4 alleles identical; thin dashed lines of increasing length, <3 alleles identical).

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