Skip directly to site content Skip directly to page options Skip directly to A-Z link Skip directly to A-Z link Skip directly to A-Z link
Volume 18, Number 2—February 2012
Dispatch

Plesiomonas shigelloides Infection, Ecuador, 2004–2008

Juan C. Escobar, Darlene Bhavnani, Gabriel Trueba, Karina Ponce, William Cevallos, and Joseph EisenbergComments to Author 
Author affiliations: University of Michigan, Ann Arbor, Michigan, USA (J. Eisenberg, D. Bhavnani); Universidad San Francisco de Quito, Quito, Ecuador (G. Trueba, W. Cevallos, K. Ponce, J.-C. Escobar)

Main Article

Figure

Case prevalence (black) and weighted community prevalence (gray) of enteric pathogens, Ecuador, 2004–2008. Identification of pathogenic Escherichia coli was based on the genes given in parentheses.

Figure. Case prevalence (black) and weighted community prevalence (white) of enteric pathogens, Ecuador, 2004–2008. Identification of pathogenic Escherichia coli was based on the genes given in parentheses. EIEC, enteroinvasive E. coli; Ipah, invasion plasmid antigen gene; ETEC, enterotoxigenic E. coli; LT, heat-labile toxin; ST, heat-stable toxin; EPEC, enteropathogenic E. coli; bfp, bundle-forming pili.

Main Article

Page created: January 18, 2012
Page updated: January 18, 2012
Page reviewed: January 18, 2012
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
file_external