Novel Epidemic Clones of Listeria monocytogenes, United States, 2011
Sara Lomonaco, Bindhu Verghese, Peter Gerner-Smidt, Cheryl Tarr, Lori Gladney, Lavin Joseph, Lee Katz, Maryann Turnsek, Michael Frace, Yi Chen, Eric Brown, Richard Meinersmann, Mark Berrang, Stephen Knabel
, and Authors
Author affiliations: Author affiliations: Università degli Studi di Torino, Turin, Italy (S. Lomonaco); Accugenix, Newark, Delaware, USA (B. Verghese); The Pennsylvania State University, University Park, Pennsylvania, USA (B. Verghese, S. Knabel); Centers for Disease Control and Prevention, Atlanta, Georgia, USA (P. Gerner-Smidt, C. Tarr, L. Gladney, L. Joseph, L. Katz, M. Turnsek, M. Frace); Food and Drug Administration, College Park, Maryland, USA (Y. Chen, E. Brown); US Department of Agriculture, Athens, Georgia, USA (R. Meinersmann, M. Berrang)
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Figure 1
Figure 1. . . Four AscI/ApaI pulsed-field gel electrophoresis (PFGE) profiles (identified at the time the research was performed) displayed by Listeria monocytogenes clinical isolates (L2624, L2625, L2626, and L2676) and isolates from food or environmental samples (LIS0072, LIS0075, LIS0077, LIS0078, and LIS0087) associated with the 2011 listeriosis outbreak traced to cantaloupe. PFGE profiles 3 and 4 differ by ≈40-kb shift in 1 band in the AscI pattern, likely related to the loss or acquisition of the comK prophage, because the size of this prophage was ≈40 kb as calculated by using the whole genome sequencing data (not shown).
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