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Volume 20, Number 6—June 2014
Research

Human Polyomavirus 9 Infection in Kidney Transplant Patients

Els van der MeijdenComments to Author , Herman F. Wunderink, Caroline S. van der Blij-de Brouwer, Hans L. Zaaijer, Joris I. Rotmans, Jan Nico Bouwes Bavinck, and Mariet C.W. Feltkamp
Author affiliations: Leiden University Medical Center, Leiden, the Netherlands (E. van der Meijden, H.F. Wunderink, C.S. van der Blij-de Brouwer, J.I. Rotmans, J.N. Bouwes Bavinck, M.C.W. Feltkamp); Sanquin Blood Supply Foundation, Amsterdam, the Netherlands (H.L. Zaaijer)

Main Article

Figure 3

Kaplan-Meier curves showing proportional increase of human polyomavirus 9 (HPyV9) DNA–positive and seropositive transplant patients during 12-month follow-up, the Netherlands. A) Cumulative HPyV9 DNA positivity (viremia) for transplant patients who were seronegative (gray) or seropositive (black) at baseline. B) Cumulative HPyV9 seropositivity for transplant patients who were nonviremic (gray) or viremic (black) at baseline.

Figure 3. Kaplan-Meier curves showing proportional increase of human polyomavirus 9 (HPyV9) DNA–positive and seropositive transplant patients during 12-month follow-up, the NetherlandsA) Cumulative HPyV9 DNA positivity (viremia) for transplant patients who were seronegative (gray) or seropositive (black) at baselineB) Cumulative HPyV9 seropositivity for transplant patients who were nonviremic (gray) or viremic (black) at baseline.

Main Article

Page created: March 28, 2014
Page updated: March 28, 2014
Page reviewed: March 28, 2014
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