Transmission of Hepatitis A Virus through Combined Liver–Small Intestine–Pancreas Transplantation
Monique A. Foster
, Lauren M. Weil, Sherry Jin, Thomas Johnson, Tonya R. Hayden-Mixson, Yury Khudyakov, Pallavi D. Annambhotla, Sridhar V. Basavaraju, Saleem Kamili, Jana M. Ritter, Noele Nelson, George Mazariegos, Michael Green, Ryan W. Himes, David T. Kuhar, Matthew J. Kuehnert, Jeffrey R. Miller, Rachel Wiseman, and Anne C. Moorman
Author affiliations: Centers for Disease Control and Prevention, Atlanta, Georgia, USA (M.A. Foster, T.R. Hayden-Mixson, Y. Khudyakov, P.D. Annambhotla, S.V. Basavaraju, S. Kamili, J.M. Ritter, N. Nelson, D.T. Kuhar, M.J. Kuehnert, J.A. Miller, A.C. Moorman); Texas Department of State Health Services, Austin, Texas, USA (L.M. Weil); Harris County Public Health and Environmental Services, Houston, Texas, USA (S. Jin); Houston Health Department, Houston (T. Johnson); Children’s Hospital of Pittsburgh of University of Pittsburgh Medical Center, Pittsburgh, Pennsylvania, USA (G. Mazariegos, M. Green); Texas Children’s Hospital, Houston, (R.W. Himes); Pennsylvania Department of Health, Harrisburg, Pennsylvania, USA (J.A. Miller); Texas Department of State Health Services, Austin (R. Wiseman)
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Figure 1
Figure 1. Polygenetic analysis of HAV isolates within the Centers for Disease Control and Prevention’s US HAV database. The genomic regions are the 315-bp long HAV VP1/P2B (viral protein 1/amino terminus of 2B). Black squares indicate isolates from the outbreak of HAV transmitted through a combined liver–small intestine–pancreas transplantation, Texas, USA, 2014–2015. Scale bar indicates nucleotide variation. HAV, hepatitis A virus.
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