Geogenomic Segregation and Temporal Trends of Human Pathogenic Escherichia coli O157:H7, Washington, USA, 2005–20141
Gillian A.M. Tarr
, Smriti Shringi, Amanda I. Phipps, Thomas E. Besser, Jonathan Mayer, Hanna N. Oltean, Jon Wakefield, Phillip I. Tarr, and Peter Rabinowitz
Author affiliations: University of Calgary, Calgary, Alberta, Canada (G.A.M. Tarr); Washington State University, Pullman, Washington, USA (S. Shringi, T.E. Besser); University of Washington, Seattle, Washington, USA (A.I. Phipps, J. Mayer, J. Wakefield, P. Rabinowitz); Washington State Department of Health, Shoreline, Washington, USA (H.N. Oltean); Washington University School of Medicine, St. Louis, Missouri, USA (P.I. Tarr)
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Figure 1
Figure 1. Escherichia coli O157:H7 lineage frequency among culture-confirmed human cases reported in Washington, USA, 2005–2014. A) Lineage Ib; B) lineage IIa; C) lineage IIb; D) rare lineages (12 different clinically rare lineages). Lineage-specific probability surfaces were determined by kernel-based estimation of spatial segregation. Darker shading indicates higher risk for that lineage. Contour lines marked 0.025 define areas in which there is a high probability of cases being caused by a given lineage, suggesting spatial segregation. Contour lines marked 0.975 define areas in which there is a low probability of cases being caused by the given lineage.
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