Westward Spread of Highly Pathogenic Avian Influenza A(H7N9) Virus among Humans, China
Qiqi Yang
1, Wei Shi
1, Lei Zhang
1, Yi Xu, Jing Xu, Shen Li, Junjun Zhang, Kan Hu, Chaofeng Ma, Xiang Zhao, Xiyan Li, Feng Liu, Xin Tong, Guogang Zhang, Pengbo Yu, Oliver G. Pybus, and Huaiyu Tian
Author affiliations: Beijing Normal University, Beijing, China (Q. Yang, X. Tong, H. Tian); Shaanxi Provincial Centre for Disease Control and Prevention, Xi’an, China (W. Shi, L. Zhang, Y. Xu, J. Xu, S. Li, F. Liu, P. Yu); Xianyang Centre for Disease Control and Prevention, Xianyang, China (J. Zhang); Baoji Centre for Disease Control and Prevention, Baoji, China (K. Hu); Xi’an Centre for Disease Control and Prevention, Xi’an (C. Ma); Chinese Center for Disease Control and Prevention, Beijing (X. Zhao, X. Li); Chinese Academy of Forestry, Beijing (G. Zhang); University of Oxford, Oxford, UK (O.G. Pybus)
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Figure 1
Figure 1. Detail of highly pathogenic avian influenza A(H7N9) viruses isolated from human and environmental sources, Shaanxi Province, China, 2016–2017, showing Shimodaira-Hasegawa-like local bootstrap support values. Amino acid changes within the hemagglutinin cleavage site are indicated on basal branches. The low pathogenicity strain A/Environment/Guangdong/15120/2016 is used as an outgroup. Colors indicate sampling locations of the H7N9 viruses obtained in this study: red, Yulin; blue, Baoji; orange, Xi’an. An expanded version of this figure showing comparisons to reference viruses is available in Technical Appendix
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Page created: May 17, 2018
Page updated: May 17, 2018
Page reviewed: May 17, 2018
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