Clonal Expansion of Macrolide-Resistant Sequence Type 3 Mycoplasma pneumoniae, South Korea
Joon Kee Lee
1, Joon Ho Lee
1, Hyunju Lee, Young Min Ahn, Byung Wook Eun, Eun Young Cho, Hwa Jin Cho, Ki Wook Yun, Hoan Jong Lee, and Eun Hwa Choi
Author affiliations: Seoul National University College of Medicine, Seoul, South Korea (J.K. Lee, J.H. Lee, H. Lee, E.Y. Cho, K.W. Yun, H.J. Lee, E.H. Choi); Seoul National University Children’s Hospital, Seoul (J.K. Lee, K.W. Yun, H.J. Lee, E.H. Choi); Kangwon National University School of Medicine, Chuncheon, South Korea (J.H. Lee); Seoul National University Bundang Hospital, Seongnam, South Korea (H. Lee); Eulji University School of Medicine, Daejeon, South Korea (Y.M. Ahn, B.W. Eun); Chungnam National University College of Medicine, Daejeon (E.Y. Cho); Chonnam National University Medical School, Gwangju, South Korea (H.J. Cho); Chonnam National University Children's Hospital, Gwangju (H.J. Cho)
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Figure 3
Figure 3. Mycoplasma pneumoniae sequence type (ST) relationship of 146 strains by eBURST analysis (http://eburst.mlst.net/), South Korea, 2000–2016. Two main CCs were defined without any singleton. ST3 and ST2 were the predicted founder of each CC. Numbers on the diagram correspond to STs. The size of each circle correlates with the number of isolates of each ST. CC, clonal complex.
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