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Volume 24, Number 9—September 2018
Dispatch

Susceptibility of White-Tailed Deer to Rift Valley Fever Virus

William C. WilsonComments to Author , In Joong Kim, Jessie D. Trujillo, Sun Young Sunwoo, Leela E. Noronha, Kinga Urbaniak, D. Scott McVey, Barbara S. Drolet, Igor Morozov, Bonto Faburay, Erin E. Schirtzinger, Tammy Koopman, Sabarish V. Indran, Velmurugan Balaraman, and Juergen A. Richt
Author affiliations: US Department of Agriculture, Manhattan, Kansas, USA (W.C. Wilson, L.E. Noronha, D.S. McVey, B.S. Drolet, E.E. Schirtzinger); Kansas State University College of Veterinary Medicine, Manhattan (I.J. Kim, J.D. Trujillo, S.Y. Sunwoo, K. Urbaniak, I. Morozov, B. Faburay, T. Koopman, S.V. Indran, V. Balaraman, J.A. Richt)

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Figure

Gross pathologic view of liver of white-tailed deer no. 44, after experimental infection with Rift Valley fever virus inoculum. The animal died at ay day 3 postinoculation; at necropsy, the liver showed severe, multifocal, hemorrhagic hepatic necrosis attributed to acute infection with Rift Valley Fever virus.

Figure. Gross pathologic view of liver of white-tailed deer no. 44 after experimental infection with Rift Valley fever virus inoculum. The animal died at day 3 postinoculation; at necropsy, the liver showed severe, multifocal, hemorrhagic hepatic necrosis attributed to acute infection with Rift Valley fever virus.

Main Article

Page created: August 15, 2018
Page updated: August 15, 2018
Page reviewed: August 15, 2018
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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