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Volume 25, Number 1—January 2019
CME ACTIVITY - Synopsis

Enterovirus A71 Infection and Neurologic Disease, Madrid, Spain, 2016

Carmen Niño Taravilla1Comments to Author , Isabel Pérez-Sebastián1, Alberto García Salido, Claudia Varela Serrano, Verónica Cantarín Extremera, Anna Duat Rodríguez, Laura López Marín, Mercedes Alonso Sanz, Olga María Suárez Traba, and Ana Serrano González
Author affiliations: Hospital Infantil Universitario Niño Jesús, Madrid, Spain

Main Article

Figure 2

Magnetic resonance images of the brain of a 2-year-old boy with enterovirus meningoencephalitis. A–C) Brain at time of diagnosis. FLAIR sequences show hyperintense lesions around ventricle IV (A), posterior region of the pons (B), and posterior region of the mesencephalon (C). D–F) Control images of cerebrum 6 months after diagnosis. FLAIR sequences show slight hyperintensity of signal around ventricle IV, lower than in the initial study (D), and complete resolution of lesions in the posterior r

Figure 2. Magnetic resonance images of the brain of a 2-year-old boy with enterovirus meningoencephalitis. A–C) Brain at time of diagnosis. FLAIR sequences show hyperintense lesions around ventricle IV (A), posterior region of the pons (B), and posterior region of the mesencephalon (C). D–F) Control images of cerebrum 6 months after diagnosis. FLAIR sequences show slight hyperintensity of signal around ventricle IV, lower than in the initial study (D), and complete resolution of lesions in the posterior region of the pons (E) and mesencephalon (F).

Main Article

1These authors contributed equally to this article.

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