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Volume 30, Number 10—October 2024
Dispatch

Autochthonous Human Babesia divergens Infection, England

Guillermo A. Zabala, Robert Lever, Xin Hui Chan, Henrietta Bristowe, Emer Kilbride, David Richards, Mark Daly, Michael Brown, Nick Johnson, Laura Eve Nabarro, Hanif Esmail, Gauri Godbole, and Peter L. ChiodiniComments to Author 
Author affiliations: Infectious Diseases Data Observatory, Oxford, UK (G.A. Zambala); Hospital for Tropical Diseases, University College Hospital, London, UK (G.A. Zabala, R. Lever, X.H. Chan, H. Bristowe, E. Kilbride, M. Brown, L.E. Nabarro, H. Esmail, G. Godbole, P.L. Chiodini); London School of Hygiene and Tropical Medicine, London (R. Lever, M. Brown, G. Godbole, P.L. Chodini); Centre for Tropical Medicine and Global Health, University of Oxford, Oxford (X.H Chan); North Devon District Hospital, Barnstaple, UK (D. Richards, M. Daly); Animal and Plant Health Agency, Surrey, UK (N. Johnson); Institute for Global Health and Medical Research Council Clinical Trials Unit, University College London, London (H. Esmail)

Main Article

Figure 1

Giemsa-stained thin blood film from a case of autochthonous human Babesia divergens infection, England. A) Stains show characteristic Maltese cross form (arrow) and erythrocytes containing 5 pyriform rings. Original magnification ×1,000. B) Absence of pigment in erythrocytes (arrow). Original magnification ×1,000.

Figure 1. Giemsa-stained thin blood film from a case of autochthonous human Babesia divergens infection, England. A) Stains show characteristic Maltese cross form (arrow) and erythrocytes containing 5 pyriform rings. Original magnification ×1,000. B) Absence of pigment in erythrocytes (arrow). Original magnification ×1,000.

Main Article

Page created: August 28, 2024
Page updated: September 23, 2024
Page reviewed: September 23, 2024
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