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Volume 9, Number 4—April 2003
Research

Hepatitis E Virus Epidemiology in Industrialized Countries

Pilar Clemente-Casares*, Sonia Pina*, Maria Buti†, Rosend Jardi†, Margarita Martín‡, Sílvia Bofill-Mas*, and Rosina Girones*Comments to Author 
Author affiliations: *University of Barcelona, Barcelona, Spain; †Hospital General Universitario Valle Hebron, Barcelona, Spain; ‡Universitat Autònoma de Barcelona, Barcelona, Spain

Main Article

Figure 1

Unrooted phylogenetic tree showing the relationship over a 123-bp fragment within open reading frame 2 between representative Hepatitis E virus strains reported in this study and other isolates from genotype I (C1, China; P, Pakistan; I, India; BCN, Barcelona, Spain; and B, Burma), genotype II (M, Mexico), genotype III (US1 and US2, United States; Sw, swine; G1 and G2, Greece; It, Italy; and Au, Austria), and genotype IV (C2 and C3, China). Strains from Barcelona, Spain, Washington, D.C., United

Figure 1. Unrooted phylogenetic tree showing the relationship over a 123-bp fragment within open reading frame 2 between representative Hepatitis E virus strains reported in this study and other isolates from genotype I (C1, China; P, Pakistan; I, India; BCN, Barcelona, Spain; and B, Burma), genotype II (M, Mexico), genotype III (US1 and US2, United States; Sw, swine; G1 and G2, Greece; It, Italy; and Au, Austria), and genotype IV (C2 and C3, China). Strains from Barcelona, Spain, Washington, D.C., United States, and Nancy, France, are in black.

Main Article

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The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
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