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Volume 10, Number 12—December 2004
Research

Nonsusceptibility of Primate Cells to Taura Syndrome Virus

Carlos R. Pantoja*Comments to Author , Solangel A. Navarro*, Jaime Naranjo*, Donald V. Lightner*, and Charles P. Gerba*
Author affiliations: *University of Arizona, Tucson, Arizona, USA

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Figure 5

Absence of reaction by in situ hybridization (ISH) to the digoxigenin (DIG)-labeled Taura syndrome virus (TSV) probes within the BGMK cells harvested at day 7 postinjection with TSV. A) No cytopathic effect suggestive of TSV infection was evident by conventional hematoxylin/eosin-phloxin (H&E) histology (H&E stain; 100x). B) Consecutive histologic section to that shown in 4A, but subjected to ISH with DIG-labeled TSV probes specific for TSV. No reaction to TSV is apparent in the challeng

Figure 5. Absence of reaction by in situ hybridization (ISH) to the digoxigenin (DIG)-labeled Taura syndrome virus (TSV) probes within the BGMK cells harvested at day 7 postinjection with TSV. A) No cytopathic effect suggestive of TSV infection was evident by conventional hematoxylin/eosin-phloxin (H&E) histology (H&E stain; 100x). B) Consecutive histologic section to that shown in 4A, but subjected to ISH with DIG-labeled TSV probes specific for TSV. No reaction to TSV is apparent in the challenged cells (Bismarck Brown counterstain; 100x).

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