Skip directly to site content Skip directly to page options Skip directly to A-Z link Skip directly to A-Z link Skip directly to A-Z link
Volume 21, Number 10—October 2015
Research

Delayed Disease Progression in Cynomolgus Macaques Infected with Ebola Virus Makona Strain

Andrea Marzi, Friederike Feldmann, Patrick W. Hanley, Dana Scott, Stephan Günther, and Heinz FeldmannComments to Author 
Author affiliations: National Institutes of Health, Hamilton, Montana, USA (A. Marzi, F. Feldmann, P.W. Hanley, D.P. Scott, H. Feldmann); Bernard-Nocht-Institute for Tropical Medicine, Hamburg, Germany (S. Günther)

Main Article

Figure 3

Serum cytokine and chemokine levels for 6 cynomolgus macaques infected with Ebola virus strains Makona or Mayinga. A) Tumor necrosis factor-α (TNF-α); B) Interleukin-6 (IL-6); C) Interferon-γ (IFN-γ); D) Soluble CD40 ligand (sCD40L); E) IL-10; and F) Monocyte chemotactic protein 1 (MCP-1). Kinetics were analyzed in serum samples of each animal collected on days of examination and time of euthanasia.

Figure 3. Serum cytokine and chemokine levels for 6 cynomolgus macaques infected with Ebola virus strains Makona or Mayinga. A) Tumor necrosis factor-α (TNF-α); B) Interleukin-6 (IL-6); C) Interferon-γ (IFN-γ); D) Soluble CD40 ligand (sCD40L); E) IL-10; and F) Monocyte chemotactic protein 1 (MCP-1). Kinetics were analyzed in serum samples of each animal collected on days of examination and time of euthanasia.

Main Article

Page created: September 22, 2015
Page updated: September 22, 2015
Page reviewed: September 22, 2015
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
file_external