Volume 21, Number 10—October 2015
Research
Delayed Disease Progression in Cynomolgus Macaques Infected with Ebola Virus Makona Strain
Figure 5
![Figure 5. Pathologic results for 6 cynomolgus macaques infected with Ebola virus strains Makona or Mayinga. Liver and spleen sections were stained with hematoxylin and eosin (H & E; top panels) and analyzed for necrosis, microthrombi, lymphocytosis, and inflammation. Sections were also stained with a polyclonal rabbit serum against Ebola virus viral protein 40 for detection of viral antigen (immunohistochemical [IHC] analysis; bottom panels). Sections from a representative animal in each group are shown. Original magnification levels: liver, ×40; spleen, ×20. Pathologic results for 6 cynomolgus macaques infected with Ebola virus strains Makona or Mayinga. Liver and spleen sections were stained with hematoxylin and eosin (H & E; top panels) and analyzed for necrosis, microthrombi, lymphocytosis, and inflammation. Sections were also stained with a polyclonal rabbit serum against Ebola virus viral protein 40 for detection of viral antigen (immunohistochemical [IHC] analysis; bottom panels). Sections from a representative animal in each group are sho](/eid/images/15-0259-F5.jpg)
Figure 5. Pathologic results for 6 cynomolgus macaques infected with Ebola virus strains Makona or Mayinga. Liver and spleen sections were stained with hematoxylin and eosin (H & E; top panels) and analyzed for necrosis, microthrombi, lymphocytosis, and inflammation. Sections were also stained with a polyclonal rabbit serum against Ebola virus viral protein 40 for detection of viral antigen (immunohistochemical [IHC] analysis; bottom panels). Sections from a representative animal in each group are shown. Original magnification levels: liver, ×40; spleen, ×20.